Color Atlas of Gross Placental Pathology by Cynthia G. Kaplan

By Cynthia G. Kaplan

This atlas is designed to assist within the cautious and thorough gross exam of the placenta through delivering an illustrated guide of exam that incorporates general adaptations, irregular findings, in addition to strange pathology. With 186 illustrations, 154 in colour, this atlas includes a wealth of vital info for pathologists analyzing the placenta. The booklet is extremely prompt for all these engaged in pathologic exam of the placenta. The textual content is concise, and the publication presents a swift reference for pathologists.

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Extra resources for Color Atlas of Gross Placental Pathology

Sample text

There is usually an associated chorioamnionitis. 31. (A) Histologically Candida funisitis shows micro abscesses just under the cord surface. These are filled with necrotic debris, in which it is difficult to identify organisms, (B) numerous fungal pseudohyphae and yeasts are present on methenamine silver stain. Infections It is more likely to cause sepsis in premature infants. 33). 32. Necrotizing funisitis represents a chronic inflammatory process in the umbilical cord, apparently infectious in origin.

Amnion nodosum is a pathologic finding, consisting of yellow-white nodules of hair and squames pressed onto the fetal surface. These nodules are not attached and can be easily removed. They may be found over the placental surface and membranes. Amnion nodosum occurs in the setting of severe oligohydramnios, and is a marker for its prior existence. 12. Another appearance of amnion nodosum is shown in this placenta which has a finely granular appearance over much of its surface. This is often much harder to recognize.

Allantoic remnants show a transitional-type epithelium and occur most often near the fetal end, between the arteries. 2). 3). It is more frequently seen with twins and velamentous cord insertions. About 20% of infants missing one artery will have other major congenital anomalies which may involve any organ system. Many are of chromosomal etiology. The abnormalities are generally apparent in the neonatal period, except for the increased incidence of inguinal hernias. The “nonmalformed” infants missing one umbilical artery are slightly growth-retarded overall and have increased perinatal mortality.

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